The actual mechanism
Bacteria in plaque metabolise sugars and starches and release acid as a by-product. That acid dissolves mineral out of the tooth surface — a process called demineralisation. Saliva pushes minerals back in between meals. A cavity forms when the acid attacks outpace the repair.
This framing matters because it explains something counterintuitive: how often you eat sugar affects decay risk more than how much. One dessert produces one acid episode. Sipping a sweetened coffee across three hours produces a near-continuous one, and saliva never gets a window to repair.
How far it has gone — and what that means for you
| Stage | What is happening | Symptoms | Treatment |
|---|---|---|---|
| Initial (white spot) | Mineral loss in enamel, surface intact | None | Possibly reversible — fluoride, plaque control |
| Enamel caries | Surface has broken; no dentin involvement | Usually none | Small filling |
| Dentin caries | Into dentin; progresses faster here | Sensitivity to cold, sweet | Filling or inlay |
| Deep / near pulp | Approaching the pulp | Lingering pain, throbbing | Deep restoration or root canal |
| Pulp involvement | Bacteria reach the nerve — pulpitis | Severe or spontaneous pain | Root canal, then crown |
Why "it doesn't hurt" means very little
Enamel contains no nerve supply at all. Pain begins only when decay reaches dentin, and reliable pain usually means it is deep. So the entire early, cheap, easily-treated phase of a cavity is silent by design.
This is also why interproximal decay — between the teeth — is so commonly missed. It is invisible in a mirror and painless for months, and it is found on a bitewing radiograph. If you have not had films taken in several years, undetected decay between teeth is the most likely thing hiding.
The sites that account for most cavities
- Molar grooves and pits — narrower than a single bristle, so a brush cannot enter them.
- Between teeth, just below the contact point. Requires floss; brushing does not reach.
- At the gumline, especially where gums have receded and softer root surface is exposed.
- Around the edges of old fillings and crowns — a gap of a few tenths of a millimetre is enough.
- Around orthodontic brackets.
- Partially erupted wisdom teeth, and the back surface of the molar in front of them.
Prevention that actually changes outcomes
- Fluoride toothpaste, and do not rinse with water afterwards. Spit, don't rinse — rinsing washes away the fluoride you just applied. This single habit change has a measurable effect.
- Reduce the frequency of sugar exposure, not just the quantity. Confine sweets to mealtimes rather than grazing.
- Clean between the teeth daily. This is where the decay you cannot see happens.
- Fissure sealants for deep molar grooves, especially in children.
- Bitewing radiographs at intervals appropriate to your risk — this is how interproximal decay gets caught while it is still small.
What treatment involves
Decayed tissue is removed and the space is restored — with composite resin for smaller cavities, or an inlay/onlay when a large portion of the tooth is gone. If decay has reached the pulp, root canal treatment becomes necessary and the tooth generally needs a crown afterwards, because a treated tooth with a large cavity is structurally weakened and prone to fracture.
Current fees for fillings, inlays, and root canal treatment are published on our English price list.